Our mission is to become a worldwide reference for education in the field for all professionals involved in the process to dissemintate knowledge & skills of Acute Cardiovascular Care
Our mission is to promote excellence in clinical diagnosis, research, technical development, and education in cardiovascular imaging in Europe.
Our mission: To promote excellence in research, practice, education and policy in cardiovascular health, primary and secondary prevention.
Our goal is to reduce the burden in cardiovascular disease in Europe through percutaneous cardiovascular interventions.
Our Mission is "to improve the quality of life of the population by reducing the impact of cardiac rhythm disturbances and reduce sudden cardiac death"
To improve quality of life and logevity, through better prevention, diagnosis and treatment of heart failure, including the establishment of networks for its management, education and research.
Working Groups goals is to stimulate and disseminate scientific knowledge in different fields of cardiology.
ESC Councils goal is to share knowledge among medical professionals practising in specific cardiology domains.
OUR MISSION: TO REDUCE THE BURDEN OF CARDIOVASCULAR DISEASE
Dr. Esther Davis
Dr. Adam Lewandowski
Dr. Paul Leeson,
Association between Sodium Intake and Change in Uric Acid, Urine Albumin Excretion, and the Risk of Developing Hypertension J.P. Forman et al. Circulation 2012; Advance online publication
Salt has traditionally been considered a symbol of friendship because of its lasting quality. However, in excess, we now know its lasting effects are not friendly to the cardiovascular system. The exact mechanisms of the association between chronic salt intake and hypertension have remained unclear but may relate to salt induced endothelial and vascular damage. J.P. Forman et al. investigated this possibility through a study of approximately 4000 normotensive individuals in their 40s from the PREVEND study. They were followed over a 6 to 9 year period and had sodium intake assessed by urinary sodium excretion, as well as measures of serum uric acid and urinary albumin excretion. They also studied the relationship of these markers to the risk of development of hypertension. They found that in those individuals without hypertension, consumption of high levels of dietary sodium was associated with increases in both serum uric acid and urinary albumin excretion. Furthermore, hypertension developed in individuals when sodium intake was associated with levels of serum uric acid and urinary albumin excretion above the highest tertile for these biomarkers. The authors argue that high serum uric acid levels reflect endothelial dysfunction and therefore, when sodium intake is sufficient to induce endothelial dysfunction and vascular damage over a prolonged period of time, hypertension develops.
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