Our mission is to become a worldwide reference for education in the field for all professionals involved in the process to dissemintate knowledge & skills of Acute Cardiovascular Care
Our mission is to promote excellence in clinical diagnosis, research, technical development, and education in cardiovascular imaging in Europe.
Our goal is to reduce the burden in cardiovascular disease in Europe through percutaneous cardiovascular interventions.
Promoting excellence in research, practice, education and policy in cardiovascular health, primary and secondary prevention.
Our Mission is "to improve the quality of life of the population by reducing the impact of cardiac rhythm disturbances and reduce sudden cardiac death"
To improve quality of life and logevity, through better prevention, diagnosis and treatment of heart failure, including the establishment of networks for its management, education and research.
Working Groups goals is to stimulate and disseminate scientific knowledge in different fields of cardiology.
ESC Councils goal is to share knowledge among medical professionals practising in specific cardiology domains.
OUR MISSION: TO REDUCE THE BURDEN OF CARDIOVASCULAR DISEASE
Panel A (top left)- Idiopathic constrictive pericarditis. Visceral epicardium is mildly inflammed, not removed since it does not interfere with myocardial function. Parietal pericardium is thickened (6-8 mm), and inestensible. Prognosis is generally very good.Panel B (top right)- Constrictive tuberculous pericarditis. Typical caseum, that in this case is sterile, negative for M.Tuberculosis. Wide areas of calcification are mechanically removed (Panel C, bottom left).Panel D (bottom right) - Constrictive pericarditis following radiotherapy. Visceral pericardium is tenacioulsy adherent to the myocardium. Removal is very difficult and technically demanding. Prognosis especially depends on the degree of myocardial involvement.
Questions:Answers will be given in the next newsletter and on the web site
1. Is pericardial thickening essential for the diagnosis of constrictive pericarditis?2. What is the risk of developing constrictive pericarditis in a patient with acute viral or idiopathic pericarditis?3. What is the risk of developing constrictive pericarditis in a patient with recurrent idiopathic pericarditis?4. How does the risk change if the patient has tuberculous pericarditis?
Different forms of constrictive pericarditis share a common physiopathology but anatomy is quite different, much more than what can be anticipated by imaging studies. In the previous image of the month, 3 different cases of constriction were depicted: idiopathic, tuberculous, and following radiotherapy.The following questions were proposed:
1. Is pericardial thickening essential for the diagnosis of constrictive pericarditis?No, pericardial thickening is not essential for the diagnosis of constrictive pericarditis. Pericardial thickness is not increased in 15-20% of patients with surgically proven constrictive pericarditis, although the histopathological appearance may be focally abnormal in all cases. When clinical, echocardiographic, or invasive hemodynamic features indicate constriction in patients with heart failure, pericardiectomy should not be denied on the basis of normal thickness as demonstrated by noninvasive imaging.
2. What is the risk of developing constrictive pericarditis in a patient with acute viral or idiopathic pericarditis? Data from a prospective color study of 500 consecutive cases of acute pericarditis of different aetiologies have been recently published. CP is a relatively rare complication of viral or idiopathic acute pericarditis (<0.5%) but, in contrast, is relatively frequent for specific aetiologies, especially bacterial.
3.What is the risk of developing constrictive pericarditis in a patient with recurrent idiopathic pericarditis? A systematic review of all publications on recurrent pericarditis from 1966 to 2006 has been published on this issue. Eight major clinical series including a total of 230 patients with idiopathic recurrent pericarditis (mean age 46 years, men/women ratio: 0.9) have been included. After a mean follow-up of 61 months, the complication rate was 3.5% cardiac tamponade and 0% constrictive pericarditis and left ventricular dysfunction. The overall life prognosis was excellent in idiopathic recurrent pericarditis and complications are uncommon. In conclusion constrictive pericarditis was never reported despite numerous recurrences, and the risk is lower than in idiopathic acute pericarditis (approximately 1%). Thus, it is important to reassure patients on their prognosis, explaining the nature of the disease, and the likely course. Therapeutic choices should take into account of the overall good outcome of these patients, including less toxic agents.4. How does the risk change if the patient has tuberculous pericarditis? Patients with tuberculous pericarditis have a substantial risk of developing constrictive pericarditis. In contemporary series from Western Europe this complication has been reported in about 20% of patients following acute tuberculous pericarditis.
European Society of Cardiology
Les Templiers2035 Route des CollesCS 80179 BIOT
06903Sophia Antipolis, FR