Our mission is to become a worldwide reference for education in the field for all professionals involved in the process to dissemintate knowledge & skills of Acute Cardiovascular Care
Our mission is to promote excellence in clinical diagnosis, research, technical development, and education in cardiovascular imaging in Europe.
Our mission: To promote excellence in research, practice, education and policy in cardiovascular health, primary and secondary prevention.
Our goal is to reduce the burden in cardiovascular disease in Europe through percutaneous cardiovascular interventions.
Our Mission is "to improve the quality of life of the population by reducing the impact of cardiac rhythm disturbances and reduce sudden cardiac death"
To improve quality of life and logevity, through better prevention, diagnosis and treatment of heart failure, including the establishment of networks for its management, education and research.
Working Groups goals is to stimulate and disseminate scientific knowledge in different fields of cardiology.
ESC Councils goal is to share knowledge among medical professionals practising in specific cardiology domains.
OUR MISSION: TO REDUCE THE BURDEN OF CARDIOVASCULAR DISEASE
A 45-year-old male visited our outpatient department due to breathless on mild exercise for 1 month. He also complained of frequent and sustained episodes of palpitations associated with shortness of breath and in the last few days he had also dyspnea at rest. He denied chest pain, syncope and fever. He was a smoker and he had a mild dyslipidaemia. He had no history of coronary artery disease, hypertension, diabetes, heavy alcohol use or illicit drug use. His family history was significant for a myocardial infarction and a sudden cardiac death, both in his father. His mother was 75 years old and lived in Romania, he hadn't any brothers or sons. There was no family history of congenital heart disease or cardiomyopathy.
Coronary angiography, which was performed to rule out ischemic heart disease, not reveal obstructive coronary artery disease (<50% stenosis). Ventriculography in the right anterior oblique (RAO) identified extensive trabeculations of the anterior, lateral, and apical regions. The overall EF was estimated at 25%. A presumptive diagnosis of left ventricular non-compaction (LVNC) was made.
Basal and contrast-enhanced cardiac magnetic resonance imaging (MRI) confirmed the presence of trabeculation and intertrabecular recesses, with a two-layered structure of the endocardium with an increased noncompacted to compacted ratio (> 2.0) in the LV lateral wall, as well as global hypokinesis and an increased LV volume, which were all compatible with LVNC.
Short episodes of clinically silent atrial fibrillation, frequent episodes of ventricular bigeminy and many premature ventricular beats were documented during telemetric monitoring.
QUESTIONSWhich of the strategies/procedures do you judge reasonable at this time?Do you think that genetic testing for LVNC is appropriate?
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