Our mission is to become a worldwide reference for education in the field for all professionals involved in the process to disseminate knowledge & skills of Acute Cardiovascular Care.
Our mission is to promote excellence in clinical diagnosis, research, technical development, and education in cardiovascular imaging in Europe.
Our mission is to promote excellence in research, practice, education and policy in cardiovascular health, primary and secondary prevention.
Our mission is to reduce the burden of cardiovascular disease through percutaneous cardiovascular interventions.
Improving the quality of life and reducing sudden cardiac death by limiting the impact of heart rhythm disturbances.
Our mission is to improve quality of life and longevity, through better prevention, diagnosis and treatment of heart failure, including the establishment of networks for its management, education and research.
The ESC Working Groups' goal is to stimulate and disseminate scientific knowledge in different fields of cardiology.
The ESC Councils' goal is to share knowledge among medical professionals practising in specific cardiology domains.
OUR MISSION: TO REDUCE THE BURDEN OF CARDIOVASCULAR DISEASE
Prof. Maurizio Galderisi,
Sudden cardiac death (SCD) is uncommon but dramatic in athletes. Cardiomyopathies (mainly hypertrophic cardiomyopathy, HCM) are the main cause of SCD in young athletes, and coronary artery disease in the older athletes. Diagnosis is difficult in athletes. Exercise capacity is not enough to exclude cardiomyopathies, but cardiac imaging evaluation can be extremely useful.
Multi-modality imaging is very important to predict SCD in HCM. Main risk factors include maximal wall thickness ≥30 mm, low ejection fraction (EF) and Global Longitudinal Strain (GLS), diastolic septum E/e’ dysfunction, left ventricular output tract (LVOT) obstruction(also provoked by exercise), late gadolinium enhancement (scar, presence and extent) and inducible myocardial ischemia.
LVEF and aortic end systolic diameter are important but questionable. GLS (>-15%) can provide important additional information in patients with normal LVEF. The rate of progression of aortic jet velocity (which is incremental to the extent of valve calcification), left ventricular hypertrophy and inappropriate increase of left ventricular mass as well as myocardial fibrosis are predictive factors in aortic stenosis. GLS , low contractile reserve and myocardial fibrosis predict cardiac events also in mitral regurgitation.
CAD is the main cause of ischemic cardiomyopathy in patients older than 35 years. LVEF alone is often used in these patients to assess long-term outcomes and the risk of SCD and pump failure, and is used to select patients for device therapy (ICD and CRT). However, LVEF has shortcomings due to variability of measurements and risk prediction on an individual basis. Myocardial scar, GLS (>-14%) and mechanical dispersion, derived from GLS, have proven incremental prognostic value over LVEF alone for predicting severe arrhythmias and SCD.
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