Our mission is to become a worldwide reference for education in the field for all professionals involved in the process to disseminate knowledge & skills of Acute Cardiovascular Care.
Our mission is to promote excellence in clinical diagnosis, research, technical development, and education in cardiovascular imaging in Europe.
Our mission is to promote excellence in research, practice, education and policy in cardiovascular health, primary and secondary prevention.
Our mission is to reduce the burden of cardiovascular disease in Europe through percutaneous cardiovascular interventions.
Our mission is to improve the quality of life of the population by reducing the impact of cardiac rhythm disturbances and reduce sudden cardiac death.
Our mission is to improve quality of life and longevity, through better prevention, diagnosis and treatment of heart failure, including the establishment of networks for its management, education and research.
The ESC Working Groups' goal is to stimulate and disseminate scientific knowledge in different fields of cardiology.
The ESC Councils' goal is to share knowledge among medical professionals practising in specific cardiology domains.
OUR MISSION: TO REDUCE THE BURDEN OF CARDIOVASCULAR DISEASE
On the left: Masson Trichrome staining On the right: SM-Aktin(brown) staining for myofibroblasts
A 12 year old girl presented with a systolic murmur when she was routinely investigated by the school doctor. For evaluating this murmur she underwent echocardiography. Here, a cardiac tumor was identified in the interventricular septum resulting in a mainly right ventricular outflow tract obstruction. The removal of the tumor (3x 2x 2 cm) by a heart surgeon was without any complications. Histopathological investigations of the tissue revealed a considerable loss of cardiomyocytes in association with a severe fibrosis (blue tissue in Masson Trichrome staining). We observed an unusual benign sinusoidal formation of structures which were CD34 negative but associated with large, focal aggregates of SM- Aktin positive myofibroblasts as demonstrated by immunohistochemistry in the right image. So far, the origin of this structural aberrance is totally unclear.
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