Our mission is to become a worldwide reference for education in the field for all professionals involved in the process to disseminate knowledge & skills of Acute Cardiovascular Care.
Our mission is to promote excellence in clinical diagnosis, research, technical development, and education in cardiovascular imaging in Europe.
Our mission is to promote excellence in research, practice, education and policy in cardiovascular health, primary and secondary prevention.
Our mission is to reduce the burden of cardiovascular disease in Europe through percutaneous cardiovascular interventions.
Our mission is to improve the quality of life of the population by reducing the impact of cardiac rhythm disturbances and reduce sudden cardiac death.
Our mission is to improve quality of life and longevity, through better prevention, diagnosis and treatment of heart failure, including the establishment of networks for its management, education and research.
The ESC Working Groups' goal is to stimulate and disseminate scientific knowledge in different fields of cardiology.
The ESC Councils' goal is to share knowledge among medical professionals practising in specific cardiology domains.
OUR MISSION: TO REDUCE THE BURDEN OF CARDIOVASCULAR DISEASE
New at Heart Failure 2016, delegates were given the opportunity to experience virtual and interactive cases on a digital simulator!
The launch of the Virtual Case Area was a great success thanks to the presenters and it's interactive audience!
We look forward to renew this at Heart Failure 2017 in Paris!
• Heart Failure clinical cases were created and presented by members of the Heart specialist Of Tomorrow (HOT) • Demonstrations were done on an interactive digital simulator• HOT presenters animated their challenging clinical cases and shared their feedback & take-home messages• Presentations took place during Coffee Breaks & the HOT/ HIT (Heart Imagers of Tomorrow) session
Patient presenting to the emergency department (ED) for worsening dyspnea since 3 weeks; dyspnea at rest (NYHA IV) and orthopnea at presentation. Given the evidence of lung congestion at chest X-ray the patient is admitted to the ICU.
The patient complains of remittent fever exacerbating in the evening hours since a couple of months, temperature rarely exceeds 38°C, anorexia and weight loss, about 5 Kg in the previous 4 to 5 weeks. Transient global amnesia about two months before, neuroimaging not performed. Current medical therapy is Amlodipine 5 mg once daily, Ramipril 5 mg once daily, Aspirin 100 mg once daily.
(before acute event):
145 / 70
O₂ saturation (%):
Watch the case presentation on SP&P
Patient initially collapsed on the street and was resuscitated. Presented Coronary artery disease (acute occlusion of the prox. LAD and RCX) with severely depressed ejection fraction, EF=30% in echocardiography. The patient presented with severe and persistent cardiogenic shock.
Patient was referred to hospital due to persistent cardiogenic shock. With positive inotropic treatment, Levosimendan, and negative balance, patient was stabilized. HF treatment was started and patient was referred to intermediate care. Despite treatment, patient did not improve and evaluation for VADs implantation and TPL was started. However, patient deteriorated and was referred to the ICU again.
Echocardiography in the context of cardiogenic shock.
22.5 (Normal weight)
139 / 80
Mickey suffered an aortic aneurysm rupture and was transferred immediately to a specialist heart and lung centre, for an emergency surgical repair of his ruptured aneurysm. Although Mickey was hemodynamically stable after surgery, 8 hours post repair his clinical condition severely deteriorated.
Patient is male, 69 years old. Recently operated to correct a ruptured aortic aneurism. Shortly after surgery, the patient was hemodynamically stable. Currently, 8 hours post repair, the patient is suffering profound peripheral vasoconstriction, higher oxygen demands, higher inotropic demands and his blood pressure is dropping acutely.
Identify hemodynamic deterioration, make a critical judgement, decide how to treat a critically ill patient.
To understand the right heart failure physiology and the optimised way of treatment.
23.9 (Normal weight)
140 / 85
© 2017 European Society of Cardiology. All rights reserved