Our mission is to become a worldwide reference for education in the field for all professionals involved in the process to dissemintate knowledge & skills of Acute Cardiovascular Care
Our mission is to promote excellence in clinical diagnosis, research, technical development, and education in cardiovascular imaging in Europe.
Our mission: To promote excellence in research, practice, education and policy in cardiovascular health, primary and secondary prevention.
Our goal is to reduce the burden in cardiovascular disease in Europe through percutaneous cardiovascular interventions.
Our Mission is "to improve the quality of life of the population by reducing the impact of cardiac rhythm disturbances and reduce sudden cardiac death"
To improve quality of life and logevity, through better prevention, diagnosis and treatment of heart failure, including the establishment of networks for its management, education and research.
Working Groups goals is to stimulate and disseminate scientific knowledge in different fields of cardiology.
ESC Councils goal is to share knowledge among medical professionals practising in specific cardiology domains.
OUR MISSION: TO REDUCE THE BURDEN OF CARDIOVASCULAR DISEASE
Dr. Jan Marek,
This symposium provided an outstanding review of our current understanding of right ventricular mechanics, including information on practical assessment and the impact on clinical practice in both congenital heart disease and pulmonary hypertension.
Beginning with a complete overview of the embryologic and myocardial architectural differences between the right and left ventricles, the session progressed to a discussion on how the right ventricle adapts to variable loading conditions. Techniques for assessing right ventricular adaptation were presented, including routine echocardiographic indices such as M-mode evaluation of ventricular septal motion, advanced speckle tracking techniques, and cardiac MRI.
These techniques provided the background to evaluate the right ventricular response to pulmonary valve implantation and to understand the subtle differences in right ventricular failure in pulmonary hypertension vs. pulmonary stenosis. The right ventricle has very different responses to different afterload states. Pulmonary hypertension induces a dilatation of the right ventricular apex and an abnormal ventricular septal shift, which results in decreased left ventricular filling, and very pronounced post-systolic shortening. Interestingly, these findings are less pronounced in the case of pulmonary stenosis, even in the case of a comparable increase in right ventricular afterload.
Comprehensive imaging of the right ventricle is paramount given the heterogeneity of congenital heart disease. Future research will be critical to further understand the different physiologic responses of the right ventricle. This goal may be accomplished by cutting edge techniques of high frame rate imaging and by larger longitudinal studies.